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Pak1 can be targeted to the membrane by Nck in response to tyrosine phosphorylation, and membrane association of Pak1 is sufficient to increase its specific activity. The mechanism whereby Pak is.
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The p21-activated kinases (PAKs) participate in cytoskeletal control networks, downstream of Rho-family GTPases. A structure of PAK1 in an autoregulated, "off" state showed that a regulatory region, N-terminal to the kinase domain, forces the latter into an inactive conformation, prevents phosphorylation of Thr423 in the activation loop, and promotes dimerization. We have now determined.
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PAKs and cancer hallmarks. PAKs are effectors of Rac/Cdc42 and play a key role in some of cancer hallmarks, including proliferative signaling, resisting cell death, activating invasion and metastasis and inducing angiogenesis. PAKs can regulate cell proliferation through the Raf/Mek pathway.
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